Pathology
Pathology/C/25
Granulomatous diseases of the lung
肺の肉芽腫性疾患
- タグ
- High-yield / ポイント
Granuloma — Recap
- Granulomatous inflammation: special form of chronic inflammation = aggregates of activated macrophages with scattered lymphocytes
- Composition: central necrosis + epithelioid cells (modified macrophages) + giant cells (fused epithelioid cells) + lymphocytes + fibroblasts
- Morphology (H&E): epithelioid cells (pink granular cytoplasm, indistinct borders); surrounded by lymphocyte collar; older granulomas surrounded by fibroblasts/CT; Langhans/multinucleated giant cells (40–50 µm; fusions of activated macrophages)
A) Tuberculosis
Causative agents
- Mycobacterium tuberculosis (mostly), M. bovis, M. africanum
- Transmitted by respiratory droplets
Clinical forms
Primary tuberculosis:
- Caseating granuloma (tubercle): caseous necrosis in center; epithelioid cells; rimmed by fibroblasts, lymphocytes, histiocytes; occasionally Langhans giant cells
- Lung lesion spreads to lymph nodes → Ranke-Ghon complex:
- Tuberculoma (parenchymal lesion) → lymphangitis → lymphadenitis
Secondary (reactivated) tuberculosis:
- Immunocompromised patients
- Affects upper lobe + apex of lung
- Symptoms: fever, hemoptysis, night sweats, chest pain, malaise
Systemic (Miliary) tuberculosis:
- Extra-pulmonary hematogenous dissemination → bacteria spread through lymphatics + bloodstream → multiple organ involvement
Diagnosis
- Acid-fast (Ziehl-Neelsen) staining, PCR, or culture
- Mycobacteria stain red; background stains blue
B) Sarcoidosis
- Multisystemic disease of unknown origin; involves lungs in 90% of cases
- Presents as bilateral hilar lymphadenopathy
Morphology
- Non-caseating epithelioid cell granulomas (non-infective)
- Giant cells with Asteroid body + Schaumann body
Other manifestations
- Skin: erythema nodosum, lupus pernio, subcutaneous nodules
- Eyes: dry eyes, blurry vision
- Lymph nodes: enlarged
- Heart: complications
- Liver and spleen: enlargement
- Joints: pain, arthritis
Diagnosis & Treatment
- Diagnosis of exclusion: culture + special stains (acid-fast) are negative
- Treatment: steroids for severe symptoms
C) Wegener Granulomatosis (Granulomatosis with Polyangiitis)
Necrotizing vasculitis characterized by a classic triad:
- Acute necrotizing granulomas of the upper and lower respiratory tract
- Necrotizing vasculitis of small- and medium-sized vessels
- Focal/diffuse necrotizing glomerulonephritis (renal vasculitis)
Pathogenesis
- Cell-mediated hypersensitivity to inhaled infectious or environmental agent
- c-ANCAs present in most cases
Clinical features
- Chronic sinusitis, mucosal ulceration of nasopharynx
- Glomerulonephritis
- Treatment: steroids
💡 High-yield: TB = caseating granuloma; Langhans giant cells; Ranke-Ghon complex; acid-fast (Ziehl-Neelsen) staining. Sarcoidosis = non-caseating granulomas; bilateral hilar LAD; Asteroid + Schaumann bodies; diagnosis of exclusion. Wegener = triad: necrotizing granulomas + vasculitis + glomerulonephritis; c-ANCA+.